Hello to all and I would like to thank all of you for your service!!! I have been reading around this site and have found a lot of useful information and I am glad there are so many of you looking out for our best interest.
I am 40% SC and was rated in 2007. I have been suffering from this condition since 2001.
20% C3-C4 and C5-C6, herniated pulposus with muscle spasms and DJD
10% Cervical Radiculopathy, right
10% Cervical Radiculopathy, left
After reading many posts with so many good suggestion of what to claim I have a few questions. I have gotten worse over the years and this year I was diagnosed with depression.
The VA psychiatrist wrote the following: Dx Axis I Depression in association witha a medical disorder, II deferred, III cervical disc disease, IV chronic pain, V GAF 60. I really don't feel like doing anything anymore and the depression medication is really not helping. I can't even sleep anymore even with the sleep medication. I have missed a lot of days from work this year to the point that I think I might even lose my job at some point if I keep this up.
I have written diagnosis from VA orthopedic doctors that rate my ROM as: Neck forward flexion was 25% of full with pain, Neck extension was 50% of full pain, Neck rotation was almost full with pain on the Rt with rotation toward the left, Neck sidebending was pain limited bilaterally. Another diagnoses read: spinal canal stenosis secondary to spondylosis of cervical spine. My most recent deep tendon reflexes exam of the biceps, brachioradialis and tricep were showing no response and that is why they sent me for the MRI last month. What can i make out of this and they seem to be down playing it. Should I be worried?
I had another MRI last month and it reads as follows:
Comparison: MRI examination from 6/11/10
Findings: There is straightening of the normal lordotic curve. There is multilevel disc space narrowing seen. The vertebral body heights are well maintained. The bone marrow signal is unremarkable with exception of Modic Type II degenerative changes within the opposing end plates at the C5-C6 level. There is normal signal within the cervical spinal cord. The prevertebral soft tissues are normal. The findings on a level by level basis are as follows:
C2-C3: No significant spinal canal stenosis or neural foraminal narrowing.
C3-C4: There is a broad-based disc bulge/osteophyte complex. Imrpovement of the previously noted left paracentral disc herniation is seen. No significant spinal canal stenosis or neural foraminal narrowing.
C4-C5: There is a broad-based disc bulge/osteophyte complex, bilateral uncovertebral joint hypertrophy. There is minimal narrowing of the spinal canal and a mild to moderate bilateral neural foraminal stenosis.
C5-C6: A mild broad-based disc bulge/osteophyte complex is noted. No significant spinal canal stenosis or neural foraminal narrowing is seen.
C6-C7: Posterior osteophytic spurring is seen. Ther is no significant spinal canal stenosis or neural foraminal narrowing.
C7-T1: No spinal canal stenosis or neural foraminal narrowing.
Impression: Imrpovement of the previously noted disc herniation at C3-C4 level is seen. Otherwise, there is no significant change in multilevel degenrative disc disease. A minimal narrowing of the spinal canal and mild to moderate bilateral neural foraminal narrowing is seen at C4-C5 level. No abnormal signal is seen in the spinal cord.
The MRI that they compared the recent one to reads as follows:
IMPRESSION: Limited study due to motion artifact. Mild - moderate multilevel degenerative spondylosis as detailed above.
REPORT: MRI cervical spine 6/11/2010..
History: Cervical pain radiating to the arms.
Sagittal and axial T2 ,sagittal T1, STIR and axial T2 * 3-D volume sequences of the cervical spine were obtained. Study is quite limited due to motion artifact. No prior studies available for comparison.
Findings: The current study reveals no evidence of acute anterior wedge compression fractures nor retropulsed fragments. Multilevel slight chronic appearing anterior stature loss felt to be degenerative in origin. There is straightening and minimal reversal of the normal cervical lordosis.
Mild- moderate multilevel degenerative spondylosis is present.
At the C3-C4 level there is disc desiccation, disc space narrowing , more so on the anterior disc margin with small central and bilateral (left slightly larger than right) disc herniation osteophytic ridge complex. Slight superior subligamentous extension of disc material over a short distance dorsal to the mid C3 segment. The uncovertebral joints are also slightly overgrown as are the facets. The disc indents the ventral surface of the thecal sac and spinal cord with mild to moderate central canal stenosis. Exit foramina appear adequate.
At the C4-C5 level, there is or significant disc desiccation and disc space narrowing. Small broad-based disc bulge osteophytic ridge complex is present and contiguous with mildly hypertrophic uncovertebral joints. Facets also mildly overgrown. The disc flattens the ventral surface of the thecal sac reaching and minimal flattening the ventral surface of the cord. Central canal is marginal to minimally narrowed . Exit foramina are slightly narrowed.
At the C5-C6 level again disc desiccation , moderate disc space narrowing , more so along the anterior disc margin. Small osteophytic ridge disc bulge complex larger on the right in the left is present. Changes are also contiguous with hypertrophic uncovertebral joints the facets are slightly overgrown. The changes compress the ventral surface of the thecal sac and spinal cord . The central canal is slightly narrowed. The exit foramina are narrowed as well.
At the C6-C7 there is marked disc desiccation and disc space narrowing. The mild Type II discogenic sclerosis along the anterior endplates noted. Small broad-based disc bulge osteophytic ridge complex is present. Uncovertebral facets are slightly overgrown. Central canal appears marginal to adequate as to the exit foramina. .
Cervicomedullary junction is unremarkable. Evaluation of cord for abnormal signal change is limited due to motion artifact.
The two reports seem to be the same findings, but the latest one is not as detailed as the one from 6/11/10. I feel like they just copied what the previous one noted. What can I make out of the MRI findings and can someone help me understand all the medical jargon.
Thank you all in advance for reading and responding.